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BONIFACINO , Tiziana

Last name: BONIFACINO

First name: Tiziana

Email: tiziana.bonifacino@unige.it

Institution: UNIGE

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2011 (v1)

Publication

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Sytemic administration of nt-1640 improves survival and motor function in the SOD1/G93A mouse model of ALS.

BONANNO, GIAMBATTISTA BONIFACINO, TIZIANA MILANESE, MARCO

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Uploaded on: April 14, 2023
2014 (v1)

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Knocking-down mGluR1 and mGluR5 in the SOD1/G93A mouse model of amyotrophic lateral sclerosis ameliorates survival and disease progression

BONANNO, GIAMBATTISTA MILANESE, MARCO BONIFACINO, TIZIANA

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Uploaded on: April 14, 2023
2013 (v1)

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Knocking-down mGluR1 and mGluR5 in the SOD1/G93A mouse model of amyotrophic lateral sclerosis ameliorates survival and disease progression

BONANNO, GIAMBATTISTA MILANESE, MARCO BONIFACINO, TIZIANA

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Uploaded on: April 14, 2023
2008 (v1)

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Effects of BND-11624 at ionotropic and metabotropic glutamatergic receptors regulating neurotransmitter release in the rat CNS

BONANNO, GIAMBATTISTA BONIFACINO, TIZIANA PITTALUGA, ANNA MARIA

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Uploaded on: April 14, 2023
2013 (v1)

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Knocking-down Group I metabotropic glutamate receptors ameliorates survival and disease progression in SOD1G93A mice.

BONANNO, GIAMBATTISTA PULITI, ALDAMARIA MILANESE, MARCO

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2009 (v1)

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Glutamate release from astrocytic gliosomes under physiological and pathological conditions

MILANESE, MARCO BONIFACINO, TIZIANA ZAPPETTINI, SIMONA

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Uploaded on: March 27, 2023
2010 (v1)

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In-vitro activation of GAT1 transporters expressed in spinal cord gliosomes stimulates glutamate release that is abnormally elevated in the SOd1/G93A(+) mouse model of amyotrophioc lateral sclerosis.

MILANESE, MARCO ZAPPETTINI, SIMONA JACCHETTI E

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Uploaded on: April 14, 2023
2016 (v1)

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Astrocyte contribution to the excessive glutamate release in the spinal cord of the SOD1G93A mouse model of amyotrophic lateral sclerosis

MILANESE, MARCO BONIFACINO, TIZIANA GALLIA, ELENA

Glutamate (Glu) excitotoxicity plays a major role in amyotrophic lateral sclerosis (ALS) and elevated extracellular Glu levels were found in patients and animal models of the disease. Reduced astrocyte uptake was suggested as the main cause for increased Glu availability. On the basis of our experiments, we postulated that abnormal...

Uploaded on: April 14, 2023
2015 (v1)

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Synaptic mechanisms underlying the excessive and precocious glutamate release in the spinal cord of sod1g93a mice.

BONIFACINO, TIZIANA MILANESE, MARCO Musazzi, L

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2015 (v1)

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Group I metabotropic glutamate receptors induce excessive glutamate release in the spinal cord of sod1g93a mice

MILANESE, MARCO BONIFACINO, TIZIANA ROSSI, PIA

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Uploaded on: April 14, 2023
2011 (v1)

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GLUTAMATE NEUROTRANSMISSION IS MODIFIED IN THE SOD1/G93A MOUSE MODEL OF ALS

USAI, CESARE MILANESE, MARCO BONIFACINO, TIZIANA

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Uploaded on: April 14, 2023
2012 (v1)

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Group I metabotropic glutamate receptors in ALS: knocking-down mGlu1 receptors ameliorates survival and disease progression in SOD1/G93A mice.

MILANESE, MARCO PULITI, ALDAMARIA BONIFACINO, TIZIANA

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Uploaded on: April 14, 2023
2013 (v1)

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G93A mutated SOD1-linked murine ALS produces abnormal exocytotic release of glutamate

USAI, CESARE MILANESE, MARCO BONIFACINO, TIZIANA

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Uploaded on: April 14, 2023
2014 (v1)

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Reducing the expression of mGlu1 and mGlu5 receptors ameliorates survival and disease progression in SOD1G93A mice

MILANESE, MARCO BONIFACINO, TIZIANA MUSANTE, ILARIA

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Uploaded on: April 14, 2023
2011 (v1)

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Abnormal exocytotic release of glutamate in a mouse model of amyotrophic lateral sclerosis

USAI, CESARE MILANESE, MARCO BONIFACINO, TIZIANA

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Uploaded on: April 14, 2023
2013 (v1)

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Partial deletion of mGluR1 receptors prolongs life span and ameliorates motor skills and biochemical and cellular parameters in a mouse model of experimental ALS

MILANESE, MARCO BONIFACINO, TIZIANA MUSANTE, ILARIA

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Uploaded on: April 14, 2023