Abnormal Glu release occurs in the spinal cord of SOD1(G93A) mice, a transgenic animal model for human ALS. Here we studied the mechanisms underlying Glu release in spinal cord nerve terminals of SOD1(G93A) mice at a pre-symptomatic disease stage (30days) and found that the basal release of Glu was more elevated in SOD1(G93A) with respect to...
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2016 (v1)PublicationUploaded on: April 14, 2023
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2015 (v1)Publication
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2013 (v1)Publication
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Uploaded on: March 27, 2023 -
2016 (v1)Publication
Clinical studies on patients with stress-related neuropsychiatric disorders reported functional and morphological changes in brain areas where glutamatergic transmission is predominant, including frontal and prefrontal areas. In line with this evidence, several preclinical works suggest that glutamate receptors are targets of both rapid and...
Uploaded on: March 27, 2023 -
2016 (v1)Publication
No description
Uploaded on: April 14, 2023 -
2016 (v1)Publication
No description
Uploaded on: April 14, 2023