VUILLEUMIER , N.

Last name: VUILLEUMIER

First name: N.

2015 (v1)

Publication

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Anti-apolipoprotein A1 autoantibodies induce tissue factor activity and expression: a role in atherothrombosis.

Pagano, S Satta, N Virzi, J

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Uploaded on: April 14, 2023
2015 (v1)

Publication

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Cd14 as a potential receptor and key regulator of anti-Apolipoproteina-1 IgG-mediated positive chronotropic effect on cardiac cells.

Mannic, T Satta, N Pagano, S

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Uploaded on: April 14, 2023
2015 (v1)

Publication

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CD14 as a mediator of the Mineralocorticoid Receptor - dependent anti-apolipoproteinA-1 IgG chronotropic effect on cardiomyocytes

Mannic, T Satta, N Pagano, S

In vitro and animal studies point to autoantibodies against apolipoproteinA-1 (anti-apoA-1 IgG) as possible mediators of cardiovascular disease involving several mechanisms, such as basal heart rate interference mediated by a mineralocorticoid receptor (MR)-dependent L-type calcium channel activation, and a direct pro-inflammatory effect...

Uploaded on: April 14, 2023
2017 (v1)

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Anti-apolipoprotein A-1 IgG predict all-cause mortality and are associated with FCRL3 polymorphisms.

Antiochos, P Marques vidal, P Virzi, J

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Uploaded on: April 14, 2023
2015 (v1)

Publication

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ApoA-1 autoantibodies increase mouse atherosclerotic plaque vulnerability, myocardial necrosis and mortality trough TLR2 and TLR4-mediated pathways.

MONTECUCCO, FABRIZIO Braunersreuther, V Burger, F

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Uploaded on: April 14, 2023
2015 (v1)

Publication

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Anti-apoA-1 auto-antibodies increase mouse atherosclerotic plaque vulnerability, myocardial necrosis and mortality triggering TLR2 and TLR4

MONTECUCCO, FABRIZIO Braunersreuther, V Burger, F

Auto-antibodies to apolipoprotein A-1 (anti-apoA-1 IgG) were shown to promote inflammation and atherogenesis, possibly through innate immune receptors signalling. Here, we aimed at investigating the role of Toll-like receptors (TLR) 2 and 4 on anti-apoA-1 IgG-induced atherosclerotic plaque vulnerability, myocardial necrosis and mortality in...

Uploaded on: April 14, 2023

VUILLEUMIER , N.

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