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2009 (v1)PublicationUploaded on: March 25, 2023
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2005 (v1)Publication
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2004 (v1)Publication
The release of [3H]d-aspartate ([3H]d-ASP) or [3H]GABA evoked by glycine and that of [3H]d-ASP or [3H]glycine evoked by GABA from spinal cord synaptosomes were studied in SOD1-G93A(+) mice, a transgenic model of amyotrophic lateral sclerosis, SOD1(+) mice and SOD1(−)/G93A(−) animals. Mutant mice were killed at advanced phase of pathology or...
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2006 (v1)Publication
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2007 (v1)PublicationEvidence that alpha7 nicotinic receptor modulates glutamate release from mouse neocortical gliosomes
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2007 (v1)Publication
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2007 (v1)Publication
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2009 (v1)Publication
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2010 (v1)Publication
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2007 (v1)Publication
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2012 (v1)Publication
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2006 (v1)Publication
Glial subcellular re-sealed particles (referred to as gliosomes here) were purified from rat cerebral cortex and investigated for their ability to release glutamate. Confocal microscopy showed that the glia-specific proteins glial fibrillary acidic protein (GFAP) and S-100, but not the neuronal proteins 95-kDa postsynaptic density protein...
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2006 (v1)Publication
Synapsins are synaptic-vesicle-associated phosphoproteins implicated in the regulation of neurotransmitter release and excitability of neuronal networks. Mutation of synapsin genes in mouse and human causes epilepsy. To understand the role of the highly conserved synapsin domain E in the dynamics of release from mammalian inhibitory neurons, we...
Uploaded on: April 14, 2023